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rabbit anti-taz polyclonal antibody  (Millipore)


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    Structured Review

    Millipore rabbit anti-taz polyclonal antibody
    Rabbit Anti Taz Polyclonal Antibody, supplied by Millipore, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/rabbit+anti-taz+polyclonal+antibody/tau++rabbit+polyclonal/pmc10873646__pnas__2310430121__sapp-54-7-21
    Average 90 stars, based on 1 article reviews
    rabbit anti-taz polyclonal antibody - by Bioz Stars, 2026-10
    90/100 stars

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    Related Articles

    Membrane:

    Article Title: Phase separation of YAP-MAML2 differentially regulates the transcriptome
    Article Snippet: The membrane was then blocked in 5% non-fat milk at room temperature for 1 hour, incubated with rabbit anti-TAZ polyclonal antibody (Millipore Sigma HPA007415, 1000x) or rabbit anti-YAP polyclonal antibody (Cell signaling 4912S, 1000X) at 4oC over-night, washed 3 times in PBST (0.1% Tween-20 in PBS), each time for 5 minutes.

    Incubation:

    Article Title: Phase separation of YAP-MAML2 differentially regulates the transcriptome
    Article Snippet: The membrane was then blocked in 5% non-fat milk at room temperature for 1 hour, incubated with rabbit anti-TAZ polyclonal antibody (Millipore Sigma HPA007415, 1000x) or rabbit anti-YAP polyclonal antibody (Cell signaling 4912S, 1000X) at 4oC over-night, washed 3 times in PBST (0.1% Tween-20 in PBS), each time for 5 minutes.



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    JTE-013 alleviated fibrosis by inhibiting the expression of RHOA, YAP, and related pathway proteins in lung tissues. ( A ): Protein–protein-interaction network constructed using the String database; ( B ): Co-expression of proteins retrieved from databases; ( C ): Immunohistochemical staining was used to detect the expression of RHOA, YAP, CTGF, and CYR61 and their OD quantitative analysis chart (scale bar = 50 μm). ( D ): Immunofluorescence staining was used to detect the expression of RHOA and their quantitative analysis chart (scale bar = 200 μm). ( E ): Immunofluorescence staining was used to detect the colocalization of YAP and α-SMA. ( F ): Western blot was used to detect the expression of RHOA, TAZ, CTGF, CYR61, and the phosphorylation of <t>Lats1</t> and YAP. Their relative levels were analyzed and compared. (** p < 0.01 vs. control group, ## p < 0.01 vs. BLM group, && p < 0.01 vs. BLM + JTE-013 group). Original Western blot available in .
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    Image Search Results


    JTE-013 alleviated fibrosis by inhibiting the expression of RHOA, YAP, and related pathway proteins in lung tissues. ( A ): Protein–protein-interaction network constructed using the String database; ( B ): Co-expression of proteins retrieved from databases; ( C ): Immunohistochemical staining was used to detect the expression of RHOA, YAP, CTGF, and CYR61 and their OD quantitative analysis chart (scale bar = 50 μm). ( D ): Immunofluorescence staining was used to detect the expression of RHOA and their quantitative analysis chart (scale bar = 200 μm). ( E ): Immunofluorescence staining was used to detect the colocalization of YAP and α-SMA. ( F ): Western blot was used to detect the expression of RHOA, TAZ, CTGF, CYR61, and the phosphorylation of Lats1 and YAP. Their relative levels were analyzed and compared. (** p < 0.01 vs. control group, ## p < 0.01 vs. BLM group, && p < 0.01 vs. BLM + JTE-013 group). Original Western blot available in .

    Journal: Pharmaceuticals

    Article Title: JTE-013 Alleviates Pulmonary Fibrosis by Affecting the RhoA/YAP Pathway and Mitochondrial Fusion/Fission

    doi: 10.3390/ph16101444

    Figure Lengend Snippet: JTE-013 alleviated fibrosis by inhibiting the expression of RHOA, YAP, and related pathway proteins in lung tissues. ( A ): Protein–protein-interaction network constructed using the String database; ( B ): Co-expression of proteins retrieved from databases; ( C ): Immunohistochemical staining was used to detect the expression of RHOA, YAP, CTGF, and CYR61 and their OD quantitative analysis chart (scale bar = 50 μm). ( D ): Immunofluorescence staining was used to detect the expression of RHOA and their quantitative analysis chart (scale bar = 200 μm). ( E ): Immunofluorescence staining was used to detect the colocalization of YAP and α-SMA. ( F ): Western blot was used to detect the expression of RHOA, TAZ, CTGF, CYR61, and the phosphorylation of Lats1 and YAP. Their relative levels were analyzed and compared. (** p < 0.01 vs. control group, ## p < 0.01 vs. BLM group, && p < 0.01 vs. BLM + JTE-013 group). Original Western blot available in .

    Article Snippet: Rabbit anti-RHOA (#bs-1180R), rabbit anti-Lats1 (#bs-2904R), rabbit anti-P-Lats1 (#bs-3246R), rabbit anti-TAZ (#bs-12367R), rabbit anti-connective tissue growth factor (CTGF) (#bs-0743R), Rabbit anti-Human Cysteine Rich Protein, Angiogenic Inducer 61 (CYR61) (#bs-1290R) were purchased from Bioss (Beijing, China).

    Techniques: Expressing, Construct, Immunohistochemistry, Staining, Immunofluorescence, Western Blot

    JTE-013 inhibited the expression of the RHOA/YAP pathway proteins and fibrosis marker proteins in alveolar epithelial MLE-12 cells. ( A ): MTT assay was used to detect the viability of MLE-12 cells treated with S1P * p < 0.05 vs. 0 μmol/L, ** p < 0.01 vs. 0 μmol/L). ( B – D ): Immunofluorescence staining was used to detect the expression of RHOA ( B ), CTGF ( C ), and YAP ( D ) (scale bar = 100 μm). ( E ): Immunofluorescence staining was used to detect the colocalization of YAP and Drp1 (scale bar = 20 μm). ( F ): Western blot was used to detect the expression of RHOA, TAZ, CTGF, CYR61, and the phosphorylation of Lats1 and YAP. ( G ): The expressions of α-SMA, COL1A1, and MMP-9 were detected by Western blot. (** p < 0.01 vs. control group, ## p < 0.01 vs. S1P group, && p < 0.01 vs. S1P + JTE-013 group). Original Western blot available in .

    Journal: Pharmaceuticals

    Article Title: JTE-013 Alleviates Pulmonary Fibrosis by Affecting the RhoA/YAP Pathway and Mitochondrial Fusion/Fission

    doi: 10.3390/ph16101444

    Figure Lengend Snippet: JTE-013 inhibited the expression of the RHOA/YAP pathway proteins and fibrosis marker proteins in alveolar epithelial MLE-12 cells. ( A ): MTT assay was used to detect the viability of MLE-12 cells treated with S1P * p < 0.05 vs. 0 μmol/L, ** p < 0.01 vs. 0 μmol/L). ( B – D ): Immunofluorescence staining was used to detect the expression of RHOA ( B ), CTGF ( C ), and YAP ( D ) (scale bar = 100 μm). ( E ): Immunofluorescence staining was used to detect the colocalization of YAP and Drp1 (scale bar = 20 μm). ( F ): Western blot was used to detect the expression of RHOA, TAZ, CTGF, CYR61, and the phosphorylation of Lats1 and YAP. ( G ): The expressions of α-SMA, COL1A1, and MMP-9 were detected by Western blot. (** p < 0.01 vs. control group, ## p < 0.01 vs. S1P group, && p < 0.01 vs. S1P + JTE-013 group). Original Western blot available in .

    Article Snippet: Rabbit anti-RHOA (#bs-1180R), rabbit anti-Lats1 (#bs-2904R), rabbit anti-P-Lats1 (#bs-3246R), rabbit anti-TAZ (#bs-12367R), rabbit anti-connective tissue growth factor (CTGF) (#bs-0743R), Rabbit anti-Human Cysteine Rich Protein, Angiogenic Inducer 61 (CYR61) (#bs-1290R) were purchased from Bioss (Beijing, China).

    Techniques: Expressing, Marker, MTT Assay, Immunofluorescence, Staining, Western Blot